See Me and Believe Me: Causality and Intersectionality in Testimonial Injustice in Healthcare

In medical settings, it is critical that all who are in need of care are correctly heard and understood. When this is not the case due to prejudices a listener has, the speaker is experiencing \emph{testimonial injustice}, which, building upon recent work, we quantify by the presence of several categories of unjust vocabulary in medical notes. In this paper, we use FCI, a causal discovery method, to study the degree to which certain demographic features could lead to marginalization (e.g., age, gender, and race) by way of contributing to testimonial injustice. To achieve this, we review physicians' notes for each patient, where we identify occurrences of unjust vocabulary, along with the demographic features present, and use causal discovery to build a Structural Causal Model (SCM) relating those demographic features to testimonial injustice. We analyze and discuss the resulting SCMs to show the interaction of these factors and how they influence the experience of injustice. Despite the potential presence of some confounding variables, we observe how one contributing feature can make a person more prone to experiencing another contributor of testimonial injustice. There is no single root of injustice and thus intersectionality cannot be ignored. These results call for considering more than singular or equalized attributes of who a person is when analyzing and improving their experiences of bias and injustice. This work is thus a first foray at using causal discovery to understand the nuanced experiences of patients in medical settings, and its insights could be used to guide design principles throughout healthcare, to build trust and promote better patient care.

Cascade-based Randomization for Inferring Causal Effects under Diffusion Interference

The presence of interference, where the outcome of an individual may depend on the treatment assignment and behavior of neighboring nodes, can lead to biased causal effect estimation. Current approaches to network experiment design focus on limiting interference through cluster-based randomization, in which clusters are identified using graph clustering, and cluster randomization dictates the node assignment to treatment and control. However, cluster-based randomization approaches perform poorly when interference propagates in cascades, whereby the response of individuals to treatment propagates to their multi-hop neighbors. When we have knowledge of the cascade seed nodes, we can leverage this interference structure to mitigate the resulting causal effect estimation bias. With this goal, we propose a cascade-based network experiment design that initiates treatment assignment from the cascade seed node and propagates the assignment to their multi-hop neighbors to limit interference during cascade growth and thereby reduce the overall causal effect estimation error. Our extensive experiments on real-world and synthetic datasets demonstrate that our proposed framework outperforms the existing state-of-the-art approaches in estimating causal effects in network data.

Bridging or Breaking: Impact of Intergroup Interactions on Religious Polarization

While exposure to diverse viewpoints may reduce polarization, it can also have a backfire effect and exacerbate polarization when the discussion is adversarial. Here, we examine the question whether intergroup interactions around important events affect polarization between majority and minority groups in social networks. We compile data on the religious identity of nearly 700,000 Indian Twitter users engaging in COVID-19-related discourse during 2020. We introduce a new measure for an individual's group conformity based on contextualized embeddings of tweet text, which helps us assess polarization between religious groups. We then use a meta-learning framework to examine heterogeneous treatment effects of intergroup interactions on an individual's group conformity in the light of communal, political, and socio-economic events. We find that for political and social events, intergroup interactions reduce polarization. This decline is weaker for individuals at the extreme who already exhibit high conformity to their group. In contrast, during communal events, intergroup interactions can increase group conformity. Finally, we decompose the differential effects across religious groups in terms of emotions and topics of discussion. The results show that the dynamics of religious polarization are sensitive to the context and have important implications for understanding the role of intergroup interactions.

Leveraging heterogeneous spillover effects in maximizing contextual bandit rewards

Recommender systems relying on contextual multi-armed bandits continuously improve relevant item recommendations by taking into account the contextual information. The objective of these bandit algorithms is to learn the best arm (i.e., best item to recommend) for each user and thus maximize the cumulative rewards from user engagement with the recommendations. However, current approaches ignore potential spillover between interacting users, where the action of one user can impact the actions and rewards of other users. Moreover, spillover may vary for different people based on their preferences and the closeness of ties to other users. This leads to heterogeneity in the spillover effects, i.e., the extent to which the action of one user can impact the action of another. Here, we propose a framework that allows contextual multi-armed bandits to account for such heterogeneous spillovers when choosing the best arm for each user. By experimenting on several real-world datasets using prominent linear and non-linear contextual bandit algorithms, we observe that our proposed method leads to significantly higher rewards than existing solutions that ignore spillover.

Mitigating Cold-start Forecasting using Cold Causal Demand Forecasting Model

Forecasting multivariate time series data, which involves predicting future values of variables over time using historical data, has significant practical applications. Although deep learning-based models have shown promise in this field, they often fail to capture the causal relationship between dependent variables, leading to less accurate forecasts. Additionally, these models cannot handle the cold-start problem in time series data, where certain variables lack historical data, posing challenges in identifying dependencies among variables. To address these limitations, we introduce the Cold Causal Demand Forecasting (CDF-cold) framework that integrates causal inference with deep learning-based models to enhance the forecasting accuracy of multivariate time series data affected by the cold-start problem. To validate the effectiveness of the proposed approach, we collect 15 multivariate time-series datasets containing the network traffic of different Google data centers. Our experiments demonstrate that the CDF-cold framework outperforms state-of-the-art forecasting models in predicting future values of multivariate time series data.

Contagion Effect Estimation Using Proximal Embeddings

Contagion effect refers to the causal effect of peers' behavior on the outcome of an individual in social networks. While prominent methods for estimating contagion effects in observational studies often assume that there are no unmeasured confounders, contagion can be confounded due to latent homophily: nodes in a homophilous network tend to have ties to peers with similar attributes and can behave similarly without influencing one another. One way to account for latent homophily is by considering proxies for the unobserved confounders. However, in the presence of high-dimensional proxies, proxy-based methods can lead to substantially biased estimation of contagion effects, as we demonstrate in this paper. To tackle this issue, we introduce the novel Proximal Embeddings (ProEmb), a framework which integrates Variational Autoencoders (VAEs) and adversarial networks to generate balanced low-dimensional representations of high-dimensional proxies for different treatment groups and identifies contagion effects in the presence of unobserved network confounders. We empirically show that our method significantly increases the accuracy of contagion effect estimation in observational network data compared to state-of-the-art methods.

Inferring Causal Effects Under Heterogeneous Peer Influence

Causal inference in networks should account for interference, which occurs when a unit's outcome is influenced by treatments or outcomes of peers. There can be heterogeneous peer influence between units when a unit's outcome is subjected to variable influence from different peers based on their attributes and relationships, or when each unit has a different susceptibility to peer influence. Existing solutions to causal inference under interference consider either homogeneous influence from peers or specific heterogeneous influence mechanisms (e.g., based on local neighborhood structure). This paper presents a methodology for estimating individual causal effects in the presence of heterogeneous peer influence due to arbitrary mechanisms. We propose a structural causal model for networks that can capture arbitrary assumptions about network structure, interference conditions, and causal dependence. We identify potential heterogeneous contexts using the causal model and propose a novel graph neural network-based estimator to estimate individual causal effects. We show that existing state-of-the-art methods for individual causal effect estimation produce biased results in the presence of heterogeneous peer influence, and that our proposed estimator is robust.

Data-Driven Estimation of Heterogeneous Treatment Effects

Estimating how a treatment affects different individuals, known as heterogeneous treatment effect estimation, is an important problem in empirical sciences. In the last few years, there has been a considerable interest in adapting machine learning algorithms to the problem of estimating heterogeneous effects from observational and experimental data. However, these algorithms often make strong assumptions about the observed features in the data and ignore the structure of the underlying causal model, which can lead to biased estimation. At the same time, the underlying causal mechanism is rarely known in real-world datasets, making it hard to take it into consideration. In this work, we provide a survey of state-of-the-art data-driven methods for heterogeneous treatment effect estimation using machine learning, broadly categorizing them as methods that focus on counterfactual prediction and methods that directly estimate the causal effect. We also provide an overview of a third category of methods which rely on structural causal models and learn the model structure from data. Our empirical evaluation under various underlying structural model mechanisms shows the advantages and deficiencies of existing estimators and of the metrics for measuring their performance.

Learning Relational Causal Models with Cycles through Relational Acyclification

In real-world phenomena which involve mutual influence or causal effects between interconnected units, equilibrium states are typically represented with cycles in graphical models. An expressive class of graphical models, \textit{relational causal models}, can represent and reason about complex dynamic systems exhibiting such cycles or feedback loops. Existing cyclic causal discovery algorithms for learning causal models from observational data assume that the data instances are independent and identically distributed which makes them unsuitable for relational causal models. At the same time, causal discovery algorithms for relational causal models assume acyclicity. In this work, we examine the necessary and sufficient conditions under which a constraint-based relational causal discovery algorithm is sound and complete for \textit{cyclic relational causal models}. We introduce \textit{relational acyclification}, an operation specifically designed for relational models that enables reasoning about the identifiability of cyclic relational causal models. We show that under the assumptions of relational acyclification and $\sigma$-faithfulness, the relational causal discovery algorithm RCD (Maier et al. 2013) is sound and complete for cyclic models. We present experimental results to support our claim.

Non-Parametric Inference of Relational Dependence

Independence testing plays a central role in statistical and causal inference from observational data. Standard independence tests assume that the data samples are independent and identically distributed (i.i.d.) but that assumption is violated in many real-world datasets and applications centered on relational systems. This work examines the problem of estimating independence in data drawn from relational systems by defining sufficient representations for the sets of observations influencing individual instances. Specifically, we define marginal and conditional independence tests for relational data by considering the kernel mean embedding as a flexible aggregation function for relational variables. We propose a consistent, non-parametric, scalable kernel test to operationalize the relational independence test for non-i.i.d. observational data under a set of structural assumptions. We empirically evaluate our proposed method on a variety of synthetic and semi-synthetic networks and demonstrate its effectiveness compared to state-of-the-art kernel-based independence tests.