Everything that can be learned about a causal structure with latent variables by observational and interventional probing schemes

What types of differences among causal structures with latent variables are impossible to distinguish by statistical data obtained by probing each visible variable? If the probing scheme is simply passive observation, then it is well-known that many different causal structures can realize the same joint probability distributions. Even for the simplest case of two visible variables, for instance, one cannot distinguish between one variable being a causal parent of the other and the two variables sharing a latent common cause. However, it is possible to distinguish between these two causal structures if we have recourse to more powerful probing schemes, such as the possibility of intervening on one of the variables and observing the other. Herein, we address the question of which causal structures remain indistinguishable even given the most informative types of probing schemes on the visible variables. We find that two causal structures remain indistinguishable if and only if they are both associated with the same mDAG structure (as defined by Evans (2016)). We also consider the question of when one causal structure dominates another in the sense that it can realize all of the joint probability distributions that can be realized by the other using a given probing scheme. (Equivalence of causal structures is the special case of mutual dominance.) Finally, we investigate to what extent one can weaken the probing schemes implemented on the visible variables and still have the same discrimination power as a maximally informative probing scheme.

Entropic Inequality Constraints from $e$-separation Relations in Directed Acyclic Graphs with Hidden Variables

Directed acyclic graphs (DAGs) with hidden variables are often used to characterize causal relations between variables in a system. When some variables are unobserved, DAGs imply a notoriously complicated set of constraints on the distribution of observed variables. In this work, we present entropic inequality constraints that are implied by $e$-separation relations in hidden variable DAGs with discrete observed variables. The constraints can intuitively be understood to follow from the fact that the capacity of variables along a causal pathway to convey information is restricted by their entropy; e.g. at the extreme case, a variable with entropy $0$ can convey no information. We show how these constraints can be used to learn about the true causal model from an observed data distribution. In addition, we propose a measure of causal influence called the minimal mediary entropy, and demonstrate that it can augment traditional measures such as the average causal effect.

Causal networks and freedom of choice in Bell's theorem

Bell's theorem is typically understood as the proof that quantum theory is incompatible with local hidden variable models. More generally, we can see the violation of a Bell inequality as witnessing the impossibility of explaining quantum correlations with classical causal models. The violation of a Bell inequality, however, does not exclude classical models where some level of measurement dependence is allowed, that is, the choice made by observers can be correlated with the source generating the systems to be measured. Here we show that the level of measurement dependence can be quantitatively upper bounded if we arrange the Bell test within a network. Furthermore, we also prove that these results can be adapted in order to derive non-linear Bell inequalities for a large class of causal networks and to identify quantumly realizable correlations which violate them.

The Inflation Technique for Causal Inference with Latent Variables

The problem of causal inference is to determine if a given probability distribution on observed variables is compatible with some causal structure. The difficult case is when the causal structure includes latent variables. We here introduce the $\textit{inflation technique}$ for tackling this problem. An inflation of a causal structure is a new causal structure that can contain multiple copies of each of the original variables, but where the ancestry of each copy mirrors that of the original. To every distribution of the observed variables that is compatible with the original causal structure, we assign a family of marginal distributions on certain subsets of the copies that are compatible with the inflated causal structure. It follows that compatibility constraints for the inflation can be translated into compatibility constraints for the original causal structure. Even if the constraints at the level of inflation are weak, such as observable statistical independences implied by disjoint causal ancestry, the translated constraints can be strong. We apply this method to derive new inequalities whose violation by a distribution witnesses that distribution's incompatibility with the causal structure (of which Bell inequalities and Pearl's instrumental inequality are prominent examples). We describe an algorithm for deriving all such inequalities for the original causal structure that follow from ancestral independences in the inflation. For three observed binary variables with pairwise common causes, it yields inequalities that are stronger in at least some aspects than those obtainable by existing methods. We also describe an algorithm that derives a weaker set of inequalities but is more efficient. Finally, we discuss which inflations are such that the inequalities one obtains from them remain valid even for quantum (and post-quantum) generalizations of the notion of a causal model.

Causal inference via algebraic geometry: feasibility tests for functional causal structures with two binary observed variables

We provide a scheme for inferring causal relations from uncontrolled statistical data based on tools from computational algebraic geometry, in particular, the computation of Groebner bases. We focus on causal structures containing just two observed variables, each of which is binary. We consider the consequences of imposing different restrictions on the number and cardinality of latent variables and of assuming different functional dependences of the observed variables on the latent ones (in particular, the noise need not be additive). We provide an inductive scheme for classifying functional causal structures into distinct observational equivalence classes. For each observational equivalence class, we provide a procedure for deriving constraints on the joint distribution that are necessary and sufficient conditions for it to arise from a model in that class. We also demonstrate how this sort of approach provides a means of determining which causal parameters are identifiable and how to solve for these. Prospects for expanding the scope of our scheme, in particular to the problem of quantum causal inference, are also discussed.

Inferring causal structure: a quantum advantage

The problem of using observed correlations to infer causal relations is relevant to a wide variety of scientific disciplines. Yet given correlations between just two classical variables, it is impossible to determine whether they arose from a causal influence of one on the other or a common cause influencing both, unless one can implement a randomized intervention. We here consider the problem of causal inference for quantum variables. We introduce causal tomography, which unifies and generalizes conventional quantum tomography schemes to provide a complete solution to the causal inference problem using a quantum analogue of a randomized trial. We furthermore show that, in contrast to the classical case, observed quantum correlations alone can sometimes provide a solution. We implement a quantum-optical experiment that allows us to control the causal relation between two optical modes, and two measurement schemes -- one with and one without randomization -- that extract this relation from the observed correlations. Our results show that entanglement and coherence, known to be central to quantum information processing, also provide a quantum advantage for causal inference.